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In addition, in PC3 mobile xenograft tumor tissues, C10 treatment inhibited tumor advancement and upregulated PKCδ. These results suggest that C10 therapy induces the PKCδ/JNK pathway, thus activating Caspase-3 and inducing the cleavage of PARP and gasdermin E to execute apoptosis and mobile-lytic pyroptosis in prostate cancer cells.This drug may